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FTR #1137 This program was recorded in one, 60-minute segment.
NB: This description contains material not contained in the original program.
Introduction: Further developing the links between biological warfare research and the Lyme Disease establishment, we review information from FTR #585.
At every turn, Lyme disease research is inextricably linked with biological warfare research. Divided into the “Steere” and “ILADS” camps, the Lyme disease research community is split between the view that the disease is “hard-to-catch, easy-to-cure” and the diametrically opposed view that the disease is very serious and produces long-term neurological disorder. The Steere camp diminishes the significance of the disease and is closely identified with biological warfare research. At the epicenter of Lyme disease research (and the Steere camp) are members of the Epidemic Intelligence Service, or EIS. EIS personnel are to be found at every bend in the road of Lyme disease research.
The Borrelia genus has long been researched as a biological warfare vector. Note that Unit 731 personnel and their files were put to work for the United States after World War II, much like the Project Paperclip scientists from Germany. ” . . . borrelia were known for their ability to adopt different forms under conditions of stress (such as exposure to antibiotics). Shedding their outer wall, (which is the target of penicillin and related drugs), they could ward off attack and continue to exist in the body. . .”
Much of the program is devoted to excerpting and analysis of a 2013 posting by Elena Cook. This discussion of “Spirochete Warfare,” in turn, makes liberal use of material from a 1944 text about Japan’s biological warfare program. This book “Japan’s Secret Weapon,” contains a great deal of information about Japanese pioneering of the use of spirochetes as biological warfare organisms.
This material is to be considered in the historical and political context of the incorporation of the key personnel and files of the notorious Japanese Unit 731 biological warfare division into the U.S. BW program after World War II.
Apparently decades ahead of their Allied counterparts, Japanese use of spirochetes encompassed a number of important points to consider:
- The Japanese understood that “cell-wall deficient spirochetes, ” “granule” and “L‑forms” had tremendous significance for biological warfare. ” . . . This WW2-era book helps to confirm what some investigating the history of Lyme disease have long suspected; that the official denial of the devastating pathogenic nature of the granule and other ‘L‑forms’(1) of Lyme-causing Borrelia, is related to their biological warfare significance. . .”
- ” . . . To put it bluntly, Newman’s book provides cogent circumstantial evidence that many Cell-wall deficient forms of Borrelia are in fact weaponized spirochetes, nurtured, cultured and optimized for aerosol delivery. . .”
- According to author Barclay Newman, a combined Japanese and Nazi biological warfare offensive against Hawaii using the spirochetal disease leptospirosis against Hawaii two or three years before the attack on Pearl Harbor: ” . . . . ‘Nazi and Japanese scientists cooperated in warfare against or with spirochetes — in Hawaii.’ (original author’s italics). What he is referring to is an exceptionally virulent outbreak of the spirochetal disease leptospirosis, also known as Weil’s disease, and known at the time in Germany as ‘slime fever’. With official reports of 44% mortality from the outbreak, Newman states: Consult the authorities, and you will find out that, very definitely, so high a mortality is attained only by Japanese strains of spirochetes of slime fever. . . .”
- According to Newman, the Japanese had concluded that spirochetes, although very close to bacteria in form, were not actually bacteria and therefore: ” . . . . a spirochete can also break itself into many tiny granules, each as small as the invisible molecule of a virus, and each capable of recreating a new spirochete. . . .”
- Again, according to Newman: ” . . . The Japanese have reported that you can increase the virulence, or killing power, of these spirals by growing them in flesh and blood, of guinea pig or man. . .” This is interesting to consider in light of the evidence of Lyme Disease as the product of biological warfare. Might some of the “tests” have had the goal of “growing” such organisms in humans? ” . . . The resistance of many spirochetes, including borrelia, to culture in vitro remains a problem for lab scientists even today. . .”
- The “granule” spirochete form was found by the Japanese to have great value for aerosolized BW applications: ” . . . Inada has reported that the Japanese know how to get virus-like, quite invisible particles or spirochete-fragments from special cultures of spirochetes of infectious jaundice. The Japanese say that such infinitesimals can be used to infect animals and men, by spraying droplets containing these spirochete-creating bits into the air, or spreading them through water, or scattering them in mud or damp soil. . . .”
- The above-mentioned leptospirosis or “slime fever” may have been used as a “softening-up” agent prior to Japanese invasions in World War II” ” . . . ‘Immediately before the Japanese invasions of China, Indo-China, the Dutch East Indies, and the Malay States, and shortly before the Japanese invasion of India and the Japanese strokes at Australia, the very first outbreaks of slime fever were reported from every one of these areas’ . . .”
- The Japanese had discovered the application of infection via multiple pathogens. This may have figured into the development of Lyme Disease as well. ” . . . Fujimori (sic) was testing out the effects of spreading two different parasites into the same guinea pig at the same time. The Japanese discovered that one parasite promotes the lethal action of the other. . . .”
- The Japanese developed with spreading spirochetal disease via spraying droplets into the eyes of targets. We wonder if Willy Burgdorfer’s possible Lyme infection from diseased Rabbit-urine may have stemmed from this technology? This is discussed below. ” . . . ‘Sometimes the Japanese think up the damnedest experiments, such as the transmission of syphilis by spraying the spirochetes into the air or into the eyes of animals or volunteers. Infection is thus accomplished. . . . if you want to speculate further about the possibilities of spirochete warfare, you can be sure that the Japanese know how to spread any spirochete disease . . . by spraying droplets laden with specially cultured spirochetes. . . .”
- Among the diseases apparently harnessed for BW use by the Japanese was African relapsing fever. Willy Burgdorfer did his graduate thesis about this tick-borne spirochetal disease and it was researched at length by his mentor Rudolf Geigy. (Geigy’s possible role as an I.G. Farben intelligence agent and Paperclip recruiter is discussed in FTR #1135. Note that some forms of Borrelia Burgdorferi–a primary causative agent of Lyme Disease–resemble the spirochete that causes relapsing fever. ” . . . Relapsing fever is caused by the Borrelia genus of bacteria, and is generally transmitted to man either by lice, or by the bite of a tick. It is worth noting, too, that recent investigations into the genetic make-up of Lyme borrelia have found some strains apparently more closely related to relapsing fever Borrelia than to Borrelia burgdorferi, long considered the only borrelia capable of causing Lyme disease. . . .”
Next, the program details Rudolf Geigy’s work on relapsing fever. We suspect that his interest in such afflictions was not as benign and altruistic as his defenders maintain. As mentioned above, Lyme Disease “discoverer” and biological warfare veteran Willy Burgdorfer did his graduate thesis on relapsing fever.
Again, as mentioned above, Willy Burgdorfer contracted what he felt was Lyme Disease after urine from an infected rabbit splashed into his eyes. We wonder if some of the techniques of using aerosolized spirochete granules might have been involved in Willy’s accidental infection? ” . . . .While he was rinsing off one of the trays in the sink, Lyme-infected rabbit urine splashed into his eyes. A few weeks later, on April 13, he noticed five Lyme bull’s-eye rashes under his armpit and on his torso. . . .”
In an unpublished manuscript, Willy Burgdorfer noted not only the persistence of Lyme Disease but its ability to remain dormant in the nervous system: “. . . . It is now clear that Borrelia burgdorferi can persist within the nervous system for years, causing progressive illness, and increasing evidence suggests also that the spirochete can remain latent there for years before producing clinical symptoms. . . .”
Lyme disease is difficult to diagnose, another factor that makes it ideal for BW use. Might the Japanese Unit 731 research into spirochetal warfare described by Barclay Newman have figured into some of the boiler-plate research that went into the development of Lyme Disease? ” . . . Lyme’s ability to evade detection on routine medical tests, its myriad presentations which can baffle doctors by mimicking 100 different diseases, its amazing abilities to evade the immune system and antibiotic treatment, would make it an attractive choice to bioweaponeers looking for an incapacitating agent. Lyme’s abilities as ‘the great imitator’ might mean that an attack could be misinterpreted as simply a rise in the incidence of different, naturally-occurring diseases. . . .”
There is experimental evidence that infection with Borrelia burgdorferi can produce the amyloid plaques symptomatic of Alzheimer’s Disease. ” . . . Here is hypothesized a truly revolutionary notion that rounded cystic forms of Borrelia burgdorferi are the root cause of the rounded structures called plaques in the Alzheimer brain. Rounded “plaques’ in high density in brain tissue are emblematic of Alzheimer’s disease (AD). . . .”
The program concludes with more experimental evidence of the production of amyloid deposits characteristic of Alzheimer’s Disease: ” . . . To determine whether an analogous host reaction to that occurring in AD could be induced by infectious agents, we exposed mammalian glial and neuronal cells in vitro to Borrelia burgdorferi spirochetes . . . Morphological changes analogous to the amyloid deposits of AD brain were observed following 2–8 weeks of exposure to the spirochetes. . . These observations indicate that, by exposure to bacteria or to their toxic products, host responses similar in nature to those observed in AD may be induced. . . .”
1. Further developing the links between biological warfare research and the Lyme Disease establishment, we review information from FTR #585.
At every turn, Lyme disease research is inextricably linked with biological warfare research. Divided into the “Steere” and “ILADS” camps, the Lyme disease research community is split between the view that the disease is “hard-to-catch, easy-to-cure” and the diametrically opposed view that the disease is very serious and produces long-term neurological disorder. The Steere camp diminishes the significance of the disease and is closely identified with biological warfare research. At the epicenter of Lyme disease research (and the Steere camp) are members of the Epidemic Intelligence Service, or EIS. EIS personnel are to be found at every bend in the road of Lyme disease research.
The Borrelia genus has long been researched as a biological warfare vector. Note that Unit 731 personnel and their files were put to work for the United States after World War II, much like the Project Paperclip scientists from Germany.
“History of Lyme disease as a Bioweapon: Lyme is a Biowarfare Issue” by Elena Cook.
. . . The Borrelia genus of bacteria, which encompasses the Borrelia burgdorferi species-group (to which Lyme disease is attributed), was studied by the infamous WW2 Japanese biowar Unit 731, who carried out horrific experiments on prisoners in Manchuria, including dissection of live human beings. [iii] Unit 731 also worked on a number of other tick-borne pathogens. After the war, the butchers of Unit 731 were shielded from prosecution by the US authorities, who wanted their expertise for the Cold War. [iv] The US government also protected and recruited German Nazi bioweaponeers under the aegis of the top-secret Operation Paperclip. . . .
2. The Borrelia genus is well suited to biological warfare research.
“History of Lyme disease as a Bioweapon: Lyme is a Biowarfare Issue” by Elena Cook.
. . . borrelia were known for their ability to adopt different forms under conditions of stress (such as exposure to antibiotics). Shedding their outer wall, (which is the target of penicillin and related drugs), they could ward off attack and continue to exist in the body. Lyme disease is not usually fatal, and it is sometimes argued that, with rapidly lethal agents like smallpox and plague available, an army would have no interest in it. However, what is important to understand here is that incapacitating or ‘non-lethal’ bioweapons are a major part of biowarfare R&D [vi], and have been for decades. . . . Military strategists understand that disabling an enemy’s soldiers can sometimes cause more damage than killing them, as large amount of resources are then tied up in caring for the casualties. An efficient incapacitating weapon dispersed over a civilian population could destroy a country’s economy and infrastructure without firing a shot. People would either be too sick to work, or too busy looking after those who were. . . .
3. Much of the program is devoted to excerpting and analysis of a 2013 posting by Elena Cook. This discussion of “Spirochete Warfare,” in turn, makes liberal use of material from a 1944 text about Japan’s biological warfare program. This book “Japan’s Secret Weapon,” contains a great deal of information about Japanese pioneering of the use of spirochetes as biological warfare organisms.
This material is to be considered in the historical and political context of the incorporation of the key personnel and files of the notorious Japanese Unit 731 biological warfare division into the U.S. BW program after World War II.
Apparently decades ahead of their Allied counterparts, Japanese use of spirochetes encompassed a number of important points to consider:
- The Japanese understood that “cell-wall deficient spirochetes, ” “granule” and “L‑forms” had tremendous significance for biological warfare. ” . . . This WW2-era book helps to confirm what some investigating the history of Lyme disease have long suspected; that the official denial of the devastating pathogenic nature of the granule and other ‘L‑forms’(1) of Lyme-causing Borrelia, is related to their biological warfare significance. . .”
- ” . . . To put it bluntly, Newman’s book provides cogent circumstantial evidence that many Cell-wall deficient forms of Borrelia are in fact weaponized spirochetes, nurtured, cultured and optimized for aerosol delivery. . .”
- According to author Barclay Newman, a combined Japanese and Nazi biological warfare offensive against Hawaii using the spirochetal disease leptospirosis against Hawaii two or three years before the attack on Pearl Harbor: ” . . . . ‘Nazi and Japanese scientists cooperated in warfare against or with spirochetes — in Hawaii.’ (original author’s italics). What he is referring to is an exceptionally virulent outbreak of the spirochetal disease leptospirosis, also known as Weil’s disease, and known at the time in Germany as ‘slime fever’. With official reports of 44% mortality from the outbreak, Newman states: Consult the authorities, and you will find out that, very definitely, so high a mortality is attained only by Japanese strains of spirochetes of slime fever. . . .”
- According to Newman, the Japanese had concluded that spirochetes, although very close to bacteria in form, were not actually bacteria and therefore: ” . . . . a spirochete can also break itself into many tiny granules, each as small as the invisible molecule of a virus, and each capable of recreating a new spirochete. . . .”
- Again, according to Newman: ” . . . The Japanese have reported that you can increase the virulence, or killing power, of these spirals by growing them in flesh and blood, of guinea pig or man. . .” This is interesting to consider in light of the evidence of Lyme Disease as the product of biological warfare. Might some of the “tests” have had the goal of “growing” such organisms in humans? ” . . . The resistance of many spirochetes, including borrelia, to culture in vitro remains a problem for lab scientists even today. . .”
- The “granule” spirochete form was found by the Japanese to have great value for aerosolized BW applications: ” . . . Inada has reported that the Japanese know how to get virus-like, quite invisible particles or spirochete-fragments from special cultures of spirochetes of infectious jaundice. The Japanese say that such infinitesimals can be used to infect animals and men, by spraying droplets containing these spirochete-creating bits into the air, or spreading them through water, or scattering them in mud or damp soil. . . .”
- The above-mentioned leptospirosis or “slime fever” may have been used as a “softening-up” agent prior to Japanese invasions in World War II” ” . . . ‘Immediately before the Japanese invasions of China, Indo-China, the Dutch East Indies, and the Malay States, and shortly before the Japanese invasion of India and the Japanese strokes at Australia, the very first outbreaks of slime fever were reported from every one of these areas’ . . .”
- The Japanese had discovered the application of infection via multiple pathogens. This may have figured into the development of Lyme Disease as well. ” . . . Fujimori (sic) was testing out the effects of spreading two different parasites into the same guinea pig at the same time. The Japanese discovered that one parasite promotes the lethal action of the other. . . .”
- The Japanese developed with spreading spirochetal disease via spraying droplets into the eyes of targets. We wonder if Willy Burgdorfer’s possible Lyme infection from diseased Rabbit-urine may have stemmed from this technology? This is discussed below. ” . . . ‘Sometimes the Japanese think up the damnedest experiments, such as the transmission of syphilis by spraying the spirochetes into the air or into the eyes of animals or volunteers. Infection is thus accomplished. . . . if you want to speculate further about the possibilities of spirochete warfare, you can be sure that the Japanese know how to spread any spirochete disease . . . by spraying droplets laden with specially cultured spirochetes. . . .”
- Among the diseases apparently harnessed for BW use by the Japanese was African relapsing fever. Willy Burgdorfer did his graduate thesis about this tick-borne spirochetal disease and it was researched at length by his mentor Rudolf Geigy. (Geigy’s possible role as an I.G. Farben intelligence agent and Paperclip recruiter is discussed in FTR #1135. Note that some forms of Borrelia Burgdorferi–a primary causative agent of Lyme Disease–resemble the spirochete that causes relapsing fever. ” . . . Relapsing fever is caused by the Borrelia genus of bacteria, and is generally transmitted to man either by lice, or by the bite of a tick. It is worth noting, too, that recent investigations into the genetic make-up of Lyme borrelia have found some strains apparently more closely related to relapsing fever Borrelia than to Borrelia burgdorferi, long considered the only borrelia capable of causing Lyme disease. . . .”
“Spirochete Warfare” by Elena Cook; 8/20/2013.
Introduction
Borrelia, the microbes which cause Lyme disease, are a sub-type of the wider biological classification of spirochetes. Now it has become apparent that the spirochetes were weaponized over 75 years ago.
That knowledge comes to us from a book published in 1944. The title of the book is “Japan’s Secret Weapon”, by Barclay Newman, a leading science writer of the time, as well as former US Navy malaria scientist.
For decades the public health agencies of the US and other NATO countries have denied the existence of virulent cell-wall deficient forms of spirochetes. The lack of a cell wall renders microbes resistant to penicillin and related antibiotics, as these work precisely by disrupting the formation of new cell walls during bacterial replication. The minute size and pleomorphic nature of these forms, in contrast to the striking spiral shape of a typical spirochete as featured in modern microbiology textbooks, made these microbes appear “invisible” — above all to those who did not wish, or did not wish others, to see them.
This WW2-era book helps to confirm what some investigating the history of Lyme disease have long suspected; that the official denial of the devastating pathogenic nature of the granule and other “L‑forms”(1) of Lyme-causing Borrelia, is related to their biological warfare significance.
To put it bluntly, Newman’s book provides cogent circumstantial evidence that many Cell-wall deficient forms of Borrelia are in fact weaponized spirochetes, nurtured, cultured and optimized for aerosol delivery.
The following essay is based on the information in Chapter IV of Newman’s book. The title of the chapter is simply “Spirochete Warfare”.
Background
For many decades it was assumed that the horrors of the Second World War did not include the use of biological weapons. Finally, in the 1980’s, thanks to the diligent efforts of historians and investigative journalists, the barbaric crimes of the Japanese Unit 731 were revealed to the general public.
Unit 731 and related units practised mass medical experimentation, including the cutting open of living human beings, who endured grotesque surgical operations without anaesthetic. Often the purpose was to observe directly the hemorrhaging and other changes in the organs of the victim — man, woman or child — as he or she died in agony from a deliberately-induced infectious disease.
As well as human experimentation, the Japanese scientists launched attacks with plague and other weapons of mass destruction, killing many thousands of Chinese and other victims. The true death toll of these atrocities is not yet known outside of classified circles.
Though the US government has long denied it, not only were they fully aware of the Japanese and Nazi biowarfare programs, but also, incredibly, after the War, they protected the architects of these programs of death from prosecution as war criminals. This was in order to recruit them for the American biological weapons program against the Soviet bloc, which they duly did.
We now know, for example, that the US allowed leading Nazi bioweaponeer Erich Traub to play a major role in setting up research at their biowarfare lab on Plum Island, a stone’s throw away from Lyme, Connecticut, where the first recorded outbreak of Lyme disease in America occurred in the 1970’s. Traub’s germ warfare knowledge was considered so important that, his Nazi past notwithstanding, he was invited to take charge of scientific research on the Island in the 1950’s.
Like Traub, Japanese biowarfare men were similarly greeted with open arms, their wartime atrocities hushed up. In return for their co-operation, the US allowed these monsters to occupy some of the most prestigious and influential posts in Japanese medicine, till their retirement decades later.
Newman’s Fear
During the War, Barclay Newman, leading science writer and former malariologist with the US Navy became aware that the Japanese were building up a program of deadly biological weaponry. Desperate to warn his countrymen of what he believed was an impending Armageddon, he wrote a book entitled “Japan’s Secret Weapon”.
At the time, the American military authorities wanted to ban his book, but later decided that to do so would call too much attention to the very issues they considered it necessary to cover up. Instead they resorted to arranging a smear campaign against the author, and unfavorable reviews dismissing Newman’s revelations as alarmist fantasy were published in the press. Today, thanks to the efforts of leading historians, we know that Newman’s fears regarding a Japanese biological program of mass destruction were soundly-based, and indeed, one of the most authoritative works on the history of biowarfare Unit 731 relies on information found in Newman’s book.(2)
“Japan’s Secret Weapon” contains no less than 28 pages on one aspect of the Japanese bioweapon program — “Spirochete Warfare”. Newman begins his chapter of the same name by lamenting the widespread disbelief, in his era, of the true devastating potential of germ warfare. He then alleges that two to three years before Pearl Harbor, “Nazi and Japanese scientists cooperated in warfare against or with spirochetes — in Hawaii.” (original author’s italics). What he is referring to is an exceptionally virulent outbreak of the spirochetal disease leptospirosis, also known as Weil’s disease, and known at the time in Germany as “slime fever”. With official reports of 44% mortality from the outbreak, Newman states: Consult the authorities, and you will find out that, very definitely, so high a mortality is attained only by Japanese strains of spirochetes of slime fever.
In his characteristic tongue-in-cheek style, Newman goes on to say the following:
Suppose you consult a spirochete specialist in his laboratory at an institute devoted to research on tropical diseases. This specialist is busy, of course. But not so busy as he ought to be or as he will be later...“It is difficult, even for an expert technician, to catch and recognize a spirochete,” the specialist informs you. “So here are some pictures from the gallery of the world’s worst rogues.”
Newman goes on to describe a picture of a typical syphilis spirochete, in a manual offered by the hypothetical spirochetologist to the reader, and then says:
“Bacterium?” you ask.
“No, according to the Japanese, who know the most about spirochetes, they are like bacteria in being low forms of plant life — that is, fungi. The Japanese claim that spirochetes are closely related to bacteria but are not bacteria, among which spiral forms are found. Like bacteria, spirochetes reproduce by splitting across the middle. But the Japanese think that a spirochete can also break itself into many tiny granules, each as small as the invisible molecule of a virus, and each capable of recreating a new spirochete. Bacteria do not seem to multiply in this odd way...The Japanese say that there is no drug effective against this spirochete.”
(Emphasis mine. It is important to bear in mind that these words were written at the dawn of the antibiotic era. Today many patients who have been lucky enough to receive a correct diagnosis of their Lyme disease have been cured, or had their symptoms alleviated, by modern therapeutic agents.)
The imaginary spirochetologist goes on to explain that much of the research on spirochetes current at that time, and even the manuals in use by US forces and the Public Health Service, are based on Japanese findings. In Newman’s scenario, the reader goes on to examine pictures of syphilis, borrelia and other spirochetes, in the US military manual, all originating from Japanese drawings:
You find out that Inada and Ito were the great investigators of the spirochetes of slime fever. When you peer closely at the dainty Japanese pictures of this spirochete, you perceive that, although at first sight it seems to be a chain of bright dots, it is really a slender thread whose spiralling gives the impression of beading. The thread is curved or hooked at one or both ends. The living spiral propels itself by rotary motion of the hook, as the Japanese discovered.
Newman then describes how the work of Hideyo Noguchi, acclaimed worldwide for his discovery of the syphilis spirochete as the cause of general paresis in 1913, was continued in Japanese labs.
Japanese technicians took a hint from Noguchi and forced the spirochete to multiply on special jellies. The Japanese have reported that you can increase the virulence, or killing power, of these spirals by growing them in flesh and blood, of guinea pig or man.
(Emphasis mine.) It is useful to remember at this point that the difficulty in culturing spirochetes using normal, ethical methods, was not just a pitfall of WW2-era technology. The resistance of many spirochetes, including borrelia, to culture in vitro remains a problem for lab scientists even today.
In Japan, vaccines for prophylaxis have long been in use. But non-Japanese workers cannot make such vaccines. None but the Japanese seems to know how to use spirochete vaccines to prevent the spread of an epidemic.
Newman goes on to discuss the Japanese discoveries of spirochetal agents of nanukayami (“seven-day fever”) and akiyami (“autumn fever”)and then, referring to one of the original discoverers of the causative agent of leptospirosis, states:
Inada has reported that the Japanese know how to get virus-like, quite invisible particles or spirochete-fragments from special cultures of spirochetes of infectious jaundice. The Japanese say that such infinitesimals can be used to infect animals and men, by spraying droplets containing these spirochete-creating bits into the air, or spreading them through water, or scattering them in mud or damp soil.
(Emphasis mine.)
Newman then discusses the prevalence of leptospirosis worldwide, and his imaginary spirochete expert notes:
“...Immediately before the Japanese invasions of China, Indo-China, the Dutch East Indies, and the Malay States, and shortly before the Japanese invasion of India and the Japanese strokes at Australia, the very first outbreaks of slime fever were reported from every one of these areas...”
After an enigmatic discussion about American and British outbreaks of leptospirosis, and the tick-borne disease tularemia in the US (the latter Newman’s scientist describes as having “somehow got in accidentally from Japan”), the reader, in the hypothetical discussion, asks about antibiotics: . . .
. . . .“They [ie the Japanese] find spirochetes especially fascinating?”
“And they never give up. In 1940, Masao Mujimori reported new successes in transmitting syphilis spirochetes from cultures grown for many years in the laboratories of Tokyo Imperial University — doubtless the very cultures started in a small way by Noguchi. Fujimori (sic) was testing out the effects of spreading two different parasites into the same guinea pig at the same time. The Japanese discovered that one parasite promotes the lethal action of the other. He demonstrated that diphtheria bacilli are more virulent when used along with syphilis ...
“Sometimes the Japanese think up the damnedest experiments, such as the transmission of syphilis by spraying the spirochetes into the air or into the eyes of animals or volunteers. Infection is thus accomplished. Japanese technicians have been not only the outstandingly successful cultivators of spirochetes and many other very deadly germs but also the sole successful mass producers of the most dangerous and horrible microbes....Some of the apparently fantastic claims of new methods of transmission by Japanese specialists have been investigated and their truth established in American laboratories years after the claims were first made. Therefore, if you want to speculate further about the possibilities of spirochete warfare, you can be sure that the Japanese know how to spread any spirochete disease — slime fever, syphilis, yaws, sodoku (3), relapsing fever — by spraying droplets laden with specially cultured spirochetes. So they do not have to drop infected fleas, rats or even leopards from planes, as suggested by popular writers.Relapsing fever is caused by the Borrelia genus of bacteria, and is generally transmitted to man either by lice, or by the bite of a tick. It is worth noting, too, that recent investigations into the genetic make-up of Lyme borrelia have found some strains apparently more closely related to relapsing fever Borrelia than to Borrelia burgdorferi, long considered the only borrelia capable of causing Lyme disease. . . .
. . . . “Hundreds of thousands, possibly millions — where?”
“In Africa today, where it is spread among tens of millions by ticks, lice and bedbugs. The spirochete of relapsing fever is almost as important a killer as malaria and trypanosomiasis, or sleeping sickness...The spirochete virulence varies widely...Only a small per cent may succumb, but in a few epidemics the mortality has attained 75 per cent. In West Africa in a recent epidemic extending through several years, probably 10 per cent of the entire population was killed off by spirochetes running wild from Morocco and Algiers down the Niger to Senegal and the French Sudan, southward to the Gold Coast and Nigeria. Perhaps a million natives died in this one epidemic...”
4a. Next, the program details Rudolf Geigy’s work on relapsing fever. We suspect that his interest in such afflictions was not as benign and altruistic as his defenders maintain.
. . . . Willy peered into the sand and saw nothing–until Geigy shook the dish: suddenly, the sand crawled to life with soft-body ticks. They looked like tiny, shriveled golden raisins with spidery black legs.
Geigy explained that in East Africa, these ticks bury themselves under the dirt floors of huts, patiently waiting for a blood meal. They spring to life when the scent organs in the tips of their front legs detect carbon dioxide from a nearby farm animal or human. Swiftly crawling onto a sleeping victim, they sink their sawtooth mouthparts into a vein and suck in warm blood for a few minutes before dropping off. During the blood meal, the tick can transmit the two potentially deadly diseases that Geigy was studying: relapsing fever and African swine fever. . . .
4b. As mentioned above, Willy Burgdorfer did his graduate thesis on relapsing fever.
“Willy Burgdorfer;” wikipedia.com.
. . . . As a research subject for his thesis he chose to study the development of the African relapsing fever spirochete, Borrelia duttonii, in its tick vector Ornithodoros moubata, and to evaluate this tick’s efficiency in transmitting spirochetes during feeding on animal hosts. . . .
5. Again, as mentioned above, Willy Burgdorfer contracted what he felt was Lyme Disease after urine from an infected rabbit splashed into his eyes. We wonder if some of the techniques of using aerosolized spirochete granules might have been involved in Willy’s accidental infection? ” . . . .While he was rinsing off one of the trays in the sink, Lyme-infected rabbit urine splashed into his eyes. A few weeks later, on April 13, he noticed five Lyme bull’s-eye rashes under his armpit and on his torso. . . .”
. . . . One Sunday in March [of 1983], he went to the lab to clean the cages of his test rabbits. The rabbits had been inoculated with cultured spirochetes, and now ticks were being allowed to feed on the animals. To prevent the ticks from escaping, the rabbits’ wire cages had been mounted over large porcelain trays filled with water. These trays had to be cleaned daily with boiling water, but because it was his lab technician’s day off, Willy had to do it. While he was rinsing off one of the trays in the sink, Lyme-infected rabbit urine splashed into his eyes. A few weeks later, on April 13, he noticed five Lyme bull’s-eye rashes under his armpit and on his torso. The family physician diagnosed the rashes as an initial reaction to a Lyme disease infection, after a test for fungi came back negative. The lab was unable to isolate an infectious organism from Willy’s blood or from a skin biopsy. Willy took a tetracycline antibiotic for twenty days before the rash disappeared. He sent a letter to Allen Steere at Yale asking for treatment advice. Always the meticulous scientist, he included a detailed sketch of his rashes, which looked very similar to those on his infected rabbits.
When the Lyme antibody test came back negative, Willy’s co-workers were divided on whether he truly had Lyme disease. Co-discoverer Alan Barbour, who had been trained as a physician, thought the rashes looked like a ringworm fungal infection. Willy insisted it was Lyme disease, explaining that the Lyme antibody test was negative only because his early dose of antibiotics may have halted the immune system reaction the test measured.
“The lesions resembled the initial skin reaction reported for Lyme disease,” he wrote. “Even though serological evaluation and attempts to isolate from blood or skin biopsy the causative agent, there appears to be no doubt at the diagnosis. Past experience has shown that patients with initial Lyme disease manifestations (lesions do not convert serologically when treated early with antibiotics.” . . . .
6. In an unpublished manuscript, Willy Burgdorfer noted not only the persistence of Lyme Disease but its ability to remain dormant in the nervous system: “. . . . It is now clear that Borrelia burgdorferi can persist within the nervous system for years, causing progressive illness, and increasing evidence suggests also that the spirochete can remain latent there for years before producing clinical symptoms. . . .”
. . . . It is now clear that Borrelia burgdorferi can persist within the nervous system for years, causing progressive illness, and increasing evidence suggests also that the spirochete can remain latent there for years before producing clinical symptoms. . . .
7. Lyme disease is difficult to diagnose, another factor that makes it ideal for BW use. Might the Japanese Unit 731 research into spirochetal warfare described by Barclay Newman have figured into some of the boiler-plate research that went into the development of Lyme Disease? ” . . . Lyme’s ability to evade detection on routine medical tests, its myriad presentations which can baffle doctors by mimicking 100 different diseases, its amazing abilities to evade the immune system and antibiotic treatment, would make it an attractive choice to bioweaponeers looking for an incapacitating agent. Lyme’s abilities as ‘the great imitator’ might mean that an attack could be misinterpreted as simply a rise in the incidence of different, naturally-occurring diseases. . . .”
“History of Lyme disease as a Bioweapon: Lyme is a Biowarfare Issue” by Elena Cook.
. . . Lyme’s ability to evade detection on routine medical tests, its myriad presentations which can baffle doctors by mimicking 100 different diseases, its amazing abilities to evade the immune system and antibiotic treatment, would make it an attractive choice to bioweaponeers looking for an incapacitating agent. Lyme’s abilities as ‘the great imitator’ might mean that an attack could be misinterpreted as simply a rise in the incidence of different, naturally-occurring diseases such as autism, MS, lupus and chronic fatigue syndrome (M.E.).
Borrelia’s inherent ability to swap outer surface proteins, which may also vary widely from strain to strain, would make the production of an effective vaccine extremely difficult. (A vaccine developed for the public by the Steere camp in collaboration with Glaxo Smithkline was pulled from the market a few years ago amid class action lawsuits [xxxvi].) Finally, the delay before the appearance of the most incapacitating symptoms would allow plenty of time for an attacker to move away from the scene, as well as preventing people in a contaminated zone from realizing they had been infected and seeking treatment. Often in the early period there is no rash, only vague flu-like or other non-specific symptoms which might be dismissed by GP’s, or ignored by the patient. . . .
8. Lyme Disease can produce symptoms that can be easily confused with other diseases.
“Spirochete Warfare” by Elena Cook; 8/20/2013.
. . . . Lyme disease, like that other spirochetal disease, syphilis, is known as a “Great Imitator”. It is believed to be able to mimic dozens of conditions, including Amyotrophic Lateral Sclerosis (Lou Gehrig’s Disease), Chronic Fatigue Syndrome or M.E., Attention Deficit Disorder, Multiple Sclerosis, Autism, Rheumatoid Arthritis, Lupus, and many more. Recent evidence has even linked it with the devastating plague of Alzheimers. (4)(5) . . .
- See “Plaques of Alzheimer’s disease originate from cysts of Borrelia burgdorferi, the Lyme disease spirochete” MacDonald AB., Med Hypotheses. 2006;67(3):592–600. Epub 2006.
- See also “Beta-amyloid deposition and Alzheimer’s type changes induced by Borrelia spirochetes”. Miklossy et al, Neurobiol Aging. 2006 Feb;27(2):228–36.
9a. There is experimental evidence that infection with Borrelia burgdorferi can produce the amyloid plaques symptomatic of Alzheimer’s Disease. ” . . . Here is hypothesized a truly revolutionary notion that rounded cystic forms of Borrelia burgdorferi are the root cause of the rounded structures called plaques in the Alzheimer brain. Rounded “plaques’ in high density in brain tissue are emblematic of Alzheimer’s disease (AD). . . .”
Summary
Here is hypothesized a truly revolutionary notion that rounded cystic forms of Borrelia burgdorferi are the root cause of the rounded structures called plaques in the Alzheimer brain. Rounded “plaques’ in high density in brain tissue are emblematic of Alzheimer’s disease (AD). Plaques may be conceptualized as rounded “pock mark-like” areas of brain tissue injury. In this century, in brain tissue of AD, plaques are Amyloid Plaques according to the most up to date textbooks. . . .
. . . . Furthermore, there is “plaque diversity” in Alzheimer’s; small, medium and large plaques parallel variable cystic diameters for Borrelia burgdorferi. . . .
. . . . In the “anatomy is destiny” model, cysts of borrelia are always round. Why then not accept roundness as a fundamental “structure determines function” argument for the answer to the mystery of why Alzheimer plaques are always round? . . .
9b. More about experimental evidence of the production of amyloid deposits characteristic of Alzheimer’s Disease: ” . . . To determine whether an analogous host reaction to that occurring in AD could be induced by infectious agents, we exposed mammalian glial and neuronal cells in vitro to Borrelia burgdorferi spirochetes . . . Morphological changes analogous to the amyloid deposits of AD brain were observed following 2–8 weeks of exposure to the spirochetes. . . These observations indicate that, by exposure to bacteria or to their toxic products, host responses similar in nature to those observed in AD may be induced. . . .”
Abstract
The pathological hallmarks of Alzheimer’s disease (AD) consist of β‑amyloid plaques and neurofibrillary tangles in affected brain areas. The processes, which drive this host reaction are unknown. To determine whether an analogous host reaction to that occurring in AD could be induced by infectious agents, we exposed mammalian glial and neuronal cells in vitro to Borrelia burgdorferi spirochetes and to the inflammatory bacterial lipopolysaccharide (LPS). Morphological changes analogous to the amyloid deposits of AD brain were observed following 2–8 weeks of exposure to the spirochetes. Increased levels of β‑amyloid precursor protein (AβPP) and hyperphosphorylated tau were also detected by Western blots of extracts of cultured cells that had been treated with spirochetes or LPS. These observations indicate that, by exposure to bacteria or to their toxic products, host responses similar in nature to those observed in AD may be induced.
The Political Implications of the UFO Phenomenon and the "ET" Myth
Well, I guess the ghosts of Kurt Blome, Erich Traub, Masaji Kitano and Shirō Ishii are coming home to roost:
https://www.bbc.com/news/world-asia-china-53303457
https://www.nytimes.com/2020/07/06/world/asia/china-bubonic-plague-inner-mongolia.html
This next article reveals that doctors may be missing signs of serious and potentially fatal brain disorders triggered by coronavirus, in mildly affected or recovering patients. UK Neurologists have documented over 40 UK Covid-19 cases with complications related to brain inflammation and delirium to nerve damage and stroke:
• 12 had inflammation of the central nervous system,
• 10 had brain disease with delirium or psychosis,
• 8 had strokes and a further eight had peripheral nerve problems, mostly diagnosed as Guillain-Barré syndrome, an immune reaction that attacks the nerves and causes paralysis (it is fatal in 5% of cases).
The full range of brain disorders caused by Covid-19 may not have been yet identified, because many patients in hospitals are too sick to examine in brain scanners or with other procedures. One concern is that the virus could leave a minority of the population with subtle brain damage that only becomes apparent in years to come. This may have also have happened in the wake of the 1918 flu pandemic, when up to a million people appeared to develop brain disease.
Warning of serious brain disorders in people with mild coronavirus symptoms
— UK neurologists publish details of mildly affected or recovering Covid-19 patients with serious or potentially fatal brain conditions
Wed 8 Jul 2020 03.44 EDT
Ian Sample Science editor Guardian, UK @iansample
Doctors may be missing signs of serious and potentially fatal brain disorders triggered by coronavirus, as they emerge in mildly affected or recovering patients, scientists have warned.
Neurologists are on Wednesday publishing details of more than 40 UK Covid-19 patients whose complications ranged from brain inflammation and delirium to nerve damage and stroke. In some cases, the neurological problem was the patient’s first and main symptom.
The cases, published in the journal Brain, revealed a rise in a life-threatening condition called acute disseminated encephalomyelitis (Adem), as the first wave of infections swept through Britain. At UCL’s Institute of Neurology, Adem cases rose from one a month before the pandemic to two or three per week in April and May. One woman, who was 59, died of the complication.
A dozen patients had inflammation of the central nervous system, 10 had brain disease with delirium or psychosis, eight had strokes and a further eight had peripheral nerve problems, mostly diagnosed as Guillain-Barré syndrome, an immune reaction that attacks the nerves and causes paralysis. It is fatal in 5% of cases.
“We’re seeing things in the way Covid-19 affects the brain that we haven’t seen before with other viruses,” said Michael Zandi, a senior author on the study and a consultant at the institute and University College London Hospitals NHS foundation trust.
“What we’ve seen with some of these Adem patients, and in other patients, is you can have severe neurology, you can be quite sick, but actually have trivial lung disease,” he added.
“Biologically, Adem has some similarities with multiple sclerosis, but it is more severe and usually happens as a one-off. Some patients are left with long-term disability, others can make a good recovery.”
The cases add to concerns over the long-term health effects of Covid-19, which have left some patients breathless and fatigued long after they have cleared the virus, and others with numbness, weakness and memory problems.
One coronavirus patient described in the paper, a 55-year-old woman with no history of psychiatric illness, began to behave oddly the day after she was discharged from hospital.
She repeatedly put her coat on and took it off again and began to hallucinate, reporting that she saw monkeys and lions in her house. She was readmitted to hospital and gradually improved on antipsychotic medication.
Another woman, aged 47, was admitted to hospital with a headache and numbness in her right hand a week after a cough and fever came on. She later became drowsy and unresponsive and required an emergency operation to remove part of her skull to relieve pressure on her swollen brain.
“We want clinicians around the world to be alert to these complications of coronavirus,” Zandi said. He urged physicians, GPs and healthcare workers with patients with cognitive symptoms, memory problems, fatigue, numbness, or weakness, to discuss the case with neurologists.
“The message is not to put that all down to the recovery, and the psychological aspects of recovery,” he said. “The brain does appear to be involved in this illness.”
The full range of brain disorders caused by Covid-19 may not have been picked up yet, because many patients in hospitals are too sick to examine in brain scanners or with other procedures. “What we really need now is better research to look at what’s really going on in the brain,” Zandi said.
One concern is that the virus could leave a minority of the population with subtle brain damage that only becomes apparent in years to come. This may have happened in the wake of the 1918 flu pandemic, when up to a million people appeared to develop brain disease.
“It’s a concern if some hidden epidemic could occur after Covid where you’re going to see delayed effects on the brain, because there could be subtle effects on the brain and slowly things happen over the coming years, but it’s far too early for us to judge now,” Zandi said.
“We hope, obviously, that that’s not going to happen, but when you’ve got such a big pandemic affecting such a vast proportion of the population it’s something we need to be alert to.”
David Strain, a senior clinical lecturer at the University of Exeter Medical School, said that only a small number of patients appeared to experience serious neurological complications and that more work was needed to understand their prevalence.
“This is very important as we start to prepare post-Covid-19 rehabilitation programs,” he said. “We’ve already seen that some people with Covid-19 may need a long rehabilitation period, both physical rehabilitation such as exercise, and brain rehabilitation. We need to understand more about the impact of this infection on the brain.”
https://www.theguardian.com/world/2020/jul/08/warning-of-serious-brain-disorders-in-people-with-mild-covid-symptoms?CMP=Share_iOSApp_Other
Most strangely, no mention can be found about Newman’s book on the internet. Perhaps Cook has the only extant copy but, just the same, this appears to be a highly successful suppression of a book that would have Orwell shaking his head in disbelief.